Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome
Identifieur interne : 000044 ( Main/Corpus ); précédent : 000043; suivant : 000045Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome
Auteurs : Robert A. Bauernfeind ; Christopher R. Wyndham ; Steven P. Swiryn ; Edwin V. Palileo ; Boris Strasberg ; Wilfred Lam ; Douglas Westveer ; Kenneth M. RosenSource :
- The American Journal of Cardiology [ 0002-9149 ] ; 1980.
Abstract
Eighty-eight patients with preexcitation were studied to determine how 30 patients with documented spontaneous paroxysmal atrial fibrillation differed from 58 patients without this arrhythmia. Inducible reentrant tachycardia was present in 23 (77 percent) of the 30 patients with, versus 28 (48 percent) of the 58 patients without, atrial fibrillation (p < 0.025). Heart disease was present in 13 (43 percent) of the 30 patients with, versus 15 (26 percent) of the 58 patients without, atrial fibrillation (not significant). Inducible reentrant tachycardia or heart disease, or both, were present in 29 (97 percent) of the 30 patients with, versus 34 (59 percent) of the 58 patients without, atrial fibrillation (p < 0.0005).Of 51 patients with inducible reentrant tachycardia, 23 patients with atrial fibrillation did not differ from 28 patients without this arrhythmia with respect to clinical features and atrial, sinus nodal, or anomalous pathway properties, or cycle length of induced reentrant tachycardia. Spontaneous degeneration of induced reentrant tachycardia to atrial fibrillation was observed in 6 (26 percent) of 23 patients with, versus none of 28 patients without, atrial fibrillation (p < 0.025).In summary, patients with preexcitation and documented spontaneous paroxysmal atrial fibrillation almost always have inducible reentrant tachycardia or heart disease, or both. It is likely that in many patients with inducible reentrant tachycardia, spontaneously occurring reentrant tachycardia relates to induction of atrial fibrillation. However, it is unclear why some patients with inducible reentrant tachycardia have atrial fibrillation and others do not. In many patients with organic heart disease, atrial fibrillation could relate to hemodynamic changes.
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DOI: 10.1016/0002-9149(81)90539-7
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Palileo</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Strasberg, Boris" sort="Strasberg, Boris" uniqKey="Strasberg B" first="Boris" last="Strasberg">Boris Strasberg</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Lam, Wilfred" sort="Lam, Wilfred" uniqKey="Lam W" first="Wilfred" last="Lam">Wilfred Lam</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Westveer, Douglas" sort="Westveer, Douglas" uniqKey="Westveer D" first="Douglas" last="Westveer">Douglas Westveer</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Rosen, Kenneth M" sort="Rosen, Kenneth M" uniqKey="Rosen K" first="Kenneth M." last="Rosen">Kenneth M. 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Bauernfeind</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Wyndham, Christopher R" sort="Wyndham, Christopher R" uniqKey="Wyndham C" first="Christopher R." last="Wyndham">Christopher R. Wyndham</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Swiryn, Steven P" sort="Swiryn, Steven P" uniqKey="Swiryn S" first="Steven P." last="Swiryn">Steven P. Swiryn</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Palileo, Edwin V" sort="Palileo, Edwin V" uniqKey="Palileo E" first="Edwin V." last="Palileo">Edwin V. Palileo</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Strasberg, Boris" sort="Strasberg, Boris" uniqKey="Strasberg B" first="Boris" last="Strasberg">Boris Strasberg</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Lam, Wilfred" sort="Lam, Wilfred" uniqKey="Lam W" first="Wilfred" last="Lam">Wilfred Lam</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Westveer, Douglas" sort="Westveer, Douglas" uniqKey="Westveer D" first="Douglas" last="Westveer">Douglas Westveer</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author><author><name sortKey="Rosen, Kenneth M" sort="Rosen, Kenneth M" uniqKey="Rosen K" first="Kenneth M." last="Rosen">Kenneth M. Rosen</name><affiliation><mods:affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="ISSN">0002-9149</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1980">1980</date><biblScope unit="volume">47</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="562">562</biblScope><biblScope unit="page" to="569">569</biblScope></imprint><idno type="ISSN">0002-9149</idno></series><idno type="istex">2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91</idno><idno type="DOI">10.1016/0002-9149(81)90539-7</idno><idno type="PII">0002-9149(81)90539-7</idno><idno type="ArticleID">81905397</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0002-9149</idno></seriesStmt></fileDesc><profileDesc><textClass></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Eighty-eight patients with preexcitation were studied to determine how 30 patients with documented spontaneous paroxysmal atrial fibrillation differed from 58 patients without this arrhythmia. Inducible reentrant tachycardia was present in 23 (77 percent) of the 30 patients with, versus 28 (48 percent) of the 58 patients without, atrial fibrillation (p < 0.025). Heart disease was present in 13 (43 percent) of the 30 patients with, versus 15 (26 percent) of the 58 patients without, atrial fibrillation (not significant). Inducible reentrant tachycardia or heart disease, or both, were present in 29 (97 percent) of the 30 patients with, versus 34 (59 percent) of the 58 patients without, atrial fibrillation (p < 0.0005).Of 51 patients with inducible reentrant tachycardia, 23 patients with atrial fibrillation did not differ from 28 patients without this arrhythmia with respect to clinical features and atrial, sinus nodal, or anomalous pathway properties, or cycle length of induced reentrant tachycardia. Spontaneous degeneration of induced reentrant tachycardia to atrial fibrillation was observed in 6 (26 percent) of 23 patients with, versus none of 28 patients without, atrial fibrillation (p < 0.025).In summary, patients with preexcitation and documented spontaneous paroxysmal atrial fibrillation almost always have inducible reentrant tachycardia or heart disease, or both. It is likely that in many patients with inducible reentrant tachycardia, spontaneously occurring reentrant tachycardia relates to induction of atrial fibrillation. However, it is unclear why some patients with inducible reentrant tachycardia have atrial fibrillation and others do not. In many patients with organic heart disease, atrial fibrillation could relate to hemodynamic changes.</div></front></TEI><istex><corpusName>elsevier</corpusName><author><json:item><name>Robert A. Bauernfeind MD, FACC</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Christopher R. Wyndham MD</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Steven P. Swiryn MD, FACC</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Edwin V. Palileo MD, FACC</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Boris Strasberg MD</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Wilfred Lam MD, FACC</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Douglas Westveer MD</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item><json:item><name>Kenneth M. Rosen MD, FACC</name><affiliations><json:string>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</json:string></affiliations></json:item></author><articleId><json:string>81905397</json:string></articleId><language><json:string>eng</json:string></language><abstract>Eighty-eight patients with preexcitation were studied to determine how 30 patients with documented spontaneous paroxysmal atrial fibrillation differed from 58 patients without this arrhythmia. Inducible reentrant tachycardia was present in 23 (77 percent) of the 30 patients with, versus 28 (48 percent) of the 58 patients without, atrial fibrillation (p > 0.025). Heart disease was present in 13 (43 percent) of the 30 patients with, versus 15 (26 percent) of the 58 patients without, atrial fibrillation (not significant). Inducible reentrant tachycardia or heart disease, or both, were present in 29 (97 percent) of the 30 patients with, versus 34 (59 percent) of the 58 patients without, atrial fibrillation (p > 0.0005).Of 51 patients with inducible reentrant tachycardia, 23 patients with atrial fibrillation did not differ from 28 patients without this arrhythmia with respect to clinical features and atrial, sinus nodal, or anomalous pathway properties, or cycle length of induced reentrant tachycardia. Spontaneous degeneration of induced reentrant tachycardia to atrial fibrillation was observed in 6 (26 percent) of 23 patients with, versus none of 28 patients without, atrial fibrillation (p > 0.025).In summary, patients with preexcitation and documented spontaneous paroxysmal atrial fibrillation almost always have inducible reentrant tachycardia or heart disease, or both. It is likely that in many patients with inducible reentrant tachycardia, spontaneously occurring reentrant tachycardia relates to induction of atrial fibrillation. However, it is unclear why some patients with inducible reentrant tachycardia have atrial fibrillation and others do not. In many patients with organic heart disease, atrial fibrillation could relate to hemodynamic changes.</abstract><qualityIndicators><score>7.671</score><pdfVersion>1.2</pdfVersion><pdfPageSize>612 x 828 pts</pdfPageSize><refBibsNative>true</refBibsNative><keywordCount>0</keywordCount><abstractCharCount>1777</abstractCharCount><pdfWordCount>4671</pdfWordCount><pdfCharCount>37269</pdfCharCount><pdfPageCount>8</pdfPageCount><abstractWordCount>255</abstractWordCount></qualityIndicators><title>Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome</title><pii><json:string>0002-9149(81)90539-7</json:string></pii><genre><json:string>research-article</json:string></genre><host><volume>47</volume><pii><json:string>S0002-9149(00)X0599-1</json:string></pii><pages><last>569</last><first>562</first></pages><issn><json:string>0002-9149</json:string></issn><issue>3</issue><genre><json:string>Journal</json:string></genre><language><json:string>unknown</json:string></language><title>The American Journal of Cardiology</title><publicationDate>1981</publicationDate></host><publicationDate>1980</publicationDate><copyrightDate>1981</copyrightDate><doi><json:string>10.1016/0002-9149(81)90539-7</json:string></doi><id>2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91</id><fulltext><json:item><original>true</original><mimetype>application/pdf</mimetype><extension>pdf</extension><uri>https://api.istex.fr/document/2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91/fulltext/pdf</uri></json:item><json:item><original>true</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91/fulltext/txt</uri></json:item><json:item><original>false</original><mimetype>application/zip</mimetype><extension>zip</extension><uri>https://api.istex.fr/document/2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91/fulltext/zip</uri></json:item><istex:fulltextTEI uri="https://api.istex.fr/document/2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91/fulltext/tei"><teiHeader><fileDesc><titleStmt><title level="a" type="main" xml:lang="en">Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome</title></titleStmt><publicationStmt><authority>ISTEX</authority><publisher>ELSEVIER</publisher><availability><p>ELSEVIER</p></availability><date>1981</date></publicationStmt><notesStmt><note>This study was supported in part by Institutional Training Grant HL 07387 and Research Grants HL 18794 and HL 23566 from the National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland and a grant from the Eleanor B. Pillsbury Resident Trust Fund.</note><note type="content">Section title: Clinical study</note></notesStmt><sourceDesc><biblStruct type="inbook"><analytic><title level="a" type="main" xml:lang="en">Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome</title><author><persName><forename type="first">Robert A.</forename><surname>Bauernfeind</surname></persName><roleName type="degree">MD, FACC</roleName><note type="biography">Address for reprints: Robert A. Bauernfeind, MD, Cardiology Section, University of Illinois Hospital, P.O. Box 6998, Chicago, Illinois 60680.</note><affiliation>Address for reprints: Robert A. Bauernfeind, MD, Cardiology Section, University of Illinois Hospital, P.O. Box 6998, Chicago, Illinois 60680.</affiliation><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Christopher R.</forename><surname>Wyndham</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Steven P.</forename><surname>Swiryn</surname></persName><roleName type="degree">MD, FACC</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Edwin V.</forename><surname>Palileo</surname></persName><roleName type="degree">MD, FACC</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Boris</forename><surname>Strasberg</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Wilfred</forename><surname>Lam</surname></persName><roleName type="degree">MD, FACC</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Douglas</forename><surname>Westveer</surname></persName><roleName type="degree">MD</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author><author><persName><forename type="first">Kenneth M.</forename><surname>Rosen</surname></persName><roleName type="degree">MD, FACC</roleName><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation></author></analytic><monogr><title level="j">The American Journal of Cardiology</title><title level="j" type="abbrev">AJC</title><idno type="pISSN">0002-9149</idno><idno type="PII">S0002-9149(00)X0599-1</idno><imprint><publisher>ELSEVIER</publisher><date type="published" when="1980"></date><biblScope unit="volume">47</biblScope><biblScope unit="issue">3</biblScope><biblScope unit="page" from="562">562</biblScope><biblScope unit="page" to="569">569</biblScope></imprint></monogr><idno type="istex">2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91</idno><idno type="DOI">10.1016/0002-9149(81)90539-7</idno><idno type="PII">0002-9149(81)90539-7</idno><idno type="ArticleID">81905397</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1981</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Eighty-eight patients with preexcitation were studied to determine how 30 patients with documented spontaneous paroxysmal atrial fibrillation differed from 58 patients without this arrhythmia. Inducible reentrant tachycardia was present in 23 (77 percent) of the 30 patients with, versus 28 (48 percent) of the 58 patients without, atrial fibrillation (p < 0.025). Heart disease was present in 13 (43 percent) of the 30 patients with, versus 15 (26 percent) of the 58 patients without, atrial fibrillation (not significant). Inducible reentrant tachycardia or heart disease, or both, were present in 29 (97 percent) of the 30 patients with, versus 34 (59 percent) of the 58 patients without, atrial fibrillation (p < 0.0005).Of 51 patients with inducible reentrant tachycardia, 23 patients with atrial fibrillation did not differ from 28 patients without this arrhythmia with respect to clinical features and atrial, sinus nodal, or anomalous pathway properties, or cycle length of induced reentrant tachycardia. Spontaneous degeneration of induced reentrant tachycardia to atrial fibrillation was observed in 6 (26 percent) of 23 patients with, versus none of 28 patients without, atrial fibrillation (p < 0.025).In summary, patients with preexcitation and documented spontaneous paroxysmal atrial fibrillation almost always have inducible reentrant tachycardia or heart disease, or both. It is likely that in many patients with inducible reentrant tachycardia, spontaneously occurring reentrant tachycardia relates to induction of atrial fibrillation. However, it is unclear why some patients with inducible reentrant tachycardia have atrial fibrillation and others do not. In many patients with organic heart disease, atrial fibrillation could relate to hemodynamic changes.</p></abstract></profileDesc><revisionDesc><change when="1980-10-24">Registration</change><change when="1980-10-13">Modified</change><change when="1980">Published</change></revisionDesc></teiHeader></istex:fulltextTEI></fulltext><metadata><istex:metadataXml wicri:clean="Elsevier, elements deleted: tail"><istex:xmlDeclaration>version="1.0" encoding="UTF-8"</istex:xmlDeclaration><istex:docType PUBLIC="-//ES//DTD journal article DTD version 4.5.2//EN//XML" URI="art452.dtd" name="istex:docType"></istex:docType><istex:document><converted-article version="4.5.2" docsubtype="fla" xml:lang="en"><item-info><jid>AJC</jid><aid>81905397</aid><ce:pii>0002-9149(81)90539-7</ce:pii><ce:doi>10.1016/0002-9149(81)90539-7</ce:doi><ce:copyright type="unknown" year="1981"></ce:copyright></item-info><head><ce:article-footnote><ce:label>☆</ce:label><ce:note-para>This study was supported in part by Institutional Training Grant HL 07387 and Research Grants HL 18794 and HL 23566 from the National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland and a grant from the Eleanor B. Pillsbury Resident Trust Fund.</ce:note-para></ce:article-footnote><ce:dochead><ce:textfn>Clinical study</ce:textfn></ce:dochead><ce:title>Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome</ce:title><ce:author-group><ce:author><ce:given-name>Robert A.</ce:given-name><ce:surname>Bauernfeind</ce:surname><ce:degrees>MD, FACC</ce:degrees><ce:cross-ref refid="COR1"><ce:sup loc="post">∗</ce:sup></ce:cross-ref></ce:author><ce:author><ce:given-name>Christopher R.</ce:given-name><ce:surname>Wyndham</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>Steven P.</ce:given-name><ce:surname>Swiryn</ce:surname><ce:degrees>MD, FACC</ce:degrees></ce:author><ce:author><ce:given-name>Edwin V.</ce:given-name><ce:surname>Palileo</ce:surname><ce:degrees>MD, FACC</ce:degrees></ce:author><ce:author><ce:given-name>Boris</ce:given-name><ce:surname>Strasberg</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>Wilfred</ce:given-name><ce:surname>Lam</ce:surname><ce:degrees>MD, FACC</ce:degrees></ce:author><ce:author><ce:given-name>Douglas</ce:given-name><ce:surname>Westveer</ce:surname><ce:degrees>MD</ce:degrees></ce:author><ce:author><ce:given-name>Kenneth M.</ce:given-name><ce:surname>Rosen</ce:surname><ce:degrees>MD, FACC</ce:degrees></ce:author><ce:affiliation><ce:textfn>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</ce:textfn></ce:affiliation><ce:correspondence id="COR1"><ce:label>∗</ce:label><ce:text>Address for reprints: Robert A. Bauernfeind, MD, Cardiology Section, University of Illinois Hospital, P.O. Box 6998, Chicago, Illinois 60680.</ce:text></ce:correspondence></ce:author-group><ce:date-received day="27" month="8" year="1980"></ce:date-received><ce:date-revised day="13" month="10" year="1980"></ce:date-revised><ce:date-accepted day="24" month="10" year="1980"></ce:date-accepted><ce:abstract class="author"><ce:section-title>Abstract</ce:section-title><ce:abstract-sec><ce:simple-para view="all" id="simple-para.0010">Eighty-eight patients with preexcitation were studied to determine how 30 patients with documented spontaneous paroxysmal atrial fibrillation differed from 58 patients without this arrhythmia. Inducible reentrant tachycardia was present in 23 (77 percent) of the 30 patients with, versus 28 (48 percent) of the 58 patients without, atrial fibrillation (p < 0.025). Heart disease was present in 13 (43 percent) of the 30 patients with, versus 15 (26 percent) of the 58 patients without, atrial fibrillation (not significant). Inducible reentrant tachycardia or heart disease, or both, were present in 29 (97 percent) of the 30 patients with, versus 34 (59 percent) of the 58 patients without, atrial fibrillation (p < 0.0005).</ce:simple-para><ce:simple-para view="all" id="simple-para.0015">Of 51 patients with inducible reentrant tachycardia, 23 patients with atrial fibrillation did not differ from 28 patients without this arrhythmia with respect to clinical features and atrial, sinus nodal, or anomalous pathway properties, or cycle length of induced reentrant tachycardia. Spontaneous degeneration of induced reentrant tachycardia to atrial fibrillation was observed in 6 (26 percent) of 23 patients with, versus none of 28 patients without, atrial fibrillation (p < 0.025).</ce:simple-para><ce:simple-para view="all" id="simple-para.0020">In summary, patients with preexcitation and documented spontaneous paroxysmal atrial fibrillation almost always have inducible reentrant tachycardia or heart disease, or both. It is likely that in many patients with inducible reentrant tachycardia, spontaneously occurring reentrant tachycardia relates to induction of atrial fibrillation. However, it is unclear why some patients with inducible reentrant tachycardia have atrial fibrillation and others do not. In many patients with organic heart disease, atrial fibrillation could relate to hemodynamic changes.</ce:simple-para></ce:abstract-sec></ce:abstract></head></converted-article></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome</title></titleInfo><titleInfo type="alternative" lang="en" contentType="CDATA"><title>Paroxysmal atrial fibrillation in the wolff-parkinson-white syndrome</title></titleInfo><name type="personal"><namePart type="given">Robert A.</namePart><namePart type="family">Bauernfeind</namePart><namePart type="termsOfAddress">MD, FACC</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><description>Address for reprints: Robert A. Bauernfeind, MD, Cardiology Section, University of Illinois Hospital, P.O. Box 6998, Chicago, Illinois 60680.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Christopher R.</namePart><namePart type="family">Wyndham</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Steven P.</namePart><namePart type="family">Swiryn</namePart><namePart type="termsOfAddress">MD, FACC</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Edwin V.</namePart><namePart type="family">Palileo</namePart><namePart type="termsOfAddress">MD, FACC</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Boris</namePart><namePart type="family">Strasberg</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Wilfred</namePart><namePart type="family">Lam</namePart><namePart type="termsOfAddress">MD, FACC</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Douglas</namePart><namePart type="family">Westveer</namePart><namePart type="termsOfAddress">MD</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">Kenneth M.</namePart><namePart type="family">Rosen</namePart><namePart type="termsOfAddress">MD, FACC</namePart><affiliation>From the Section of Cardiology, Department of Medicine, Abraham Lincoln School of Medicine, University of Illinois College of Medicine, Chicago, Illinois USA</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="research-article" displayLabel="Full-length article"></genre><originInfo><publisher>ELSEVIER</publisher><dateIssued encoding="w3cdtf">1980</dateIssued><dateValid encoding="w3cdtf">1980-10-24</dateValid><dateModified encoding="w3cdtf">1980-10-13</dateModified><copyrightDate encoding="w3cdtf">1981</copyrightDate></originInfo><language><languageTerm type="code" authority="iso639-2b">eng</languageTerm><languageTerm type="code" authority="rfc3066">en</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType></physicalDescription><abstract lang="en">Eighty-eight patients with preexcitation were studied to determine how 30 patients with documented spontaneous paroxysmal atrial fibrillation differed from 58 patients without this arrhythmia. Inducible reentrant tachycardia was present in 23 (77 percent) of the 30 patients with, versus 28 (48 percent) of the 58 patients without, atrial fibrillation (p < 0.025). Heart disease was present in 13 (43 percent) of the 30 patients with, versus 15 (26 percent) of the 58 patients without, atrial fibrillation (not significant). Inducible reentrant tachycardia or heart disease, or both, were present in 29 (97 percent) of the 30 patients with, versus 34 (59 percent) of the 58 patients without, atrial fibrillation (p < 0.0005).Of 51 patients with inducible reentrant tachycardia, 23 patients with atrial fibrillation did not differ from 28 patients without this arrhythmia with respect to clinical features and atrial, sinus nodal, or anomalous pathway properties, or cycle length of induced reentrant tachycardia. Spontaneous degeneration of induced reentrant tachycardia to atrial fibrillation was observed in 6 (26 percent) of 23 patients with, versus none of 28 patients without, atrial fibrillation (p < 0.025).In summary, patients with preexcitation and documented spontaneous paroxysmal atrial fibrillation almost always have inducible reentrant tachycardia or heart disease, or both. It is likely that in many patients with inducible reentrant tachycardia, spontaneously occurring reentrant tachycardia relates to induction of atrial fibrillation. However, it is unclear why some patients with inducible reentrant tachycardia have atrial fibrillation and others do not. In many patients with organic heart disease, atrial fibrillation could relate to hemodynamic changes.</abstract><note>This study was supported in part by Institutional Training Grant HL 07387 and Research Grants HL 18794 and HL 23566 from the National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland and a grant from the Eleanor B. Pillsbury Resident Trust Fund.</note><note type="content">Section title: Clinical study</note><relatedItem type="host"><titleInfo><title>The American Journal of Cardiology</title></titleInfo><titleInfo type="abbreviated"><title>AJC</title></titleInfo><genre type="Journal">journal</genre><originInfo><dateIssued encoding="w3cdtf">198103</dateIssued></originInfo><identifier type="ISSN">0002-9149</identifier><identifier type="PII">S0002-9149(00)X0599-1</identifier><part><date>198103</date><detail type="volume"><number>47</number><caption>vol.</caption></detail><detail type="issue"><number>3</number><caption>no.</caption></detail><extent unit="issue pages"><start>A101</start><end>A108</end></extent><extent unit="issue pages"><start>A109</start><end>A114</end></extent><extent unit="issue pages"><start>A115</start><end>A118</end></extent><extent unit="issue pages"><start>A119</start><end>A134</end></extent><extent unit="issue pages"><start>A135</start><end>A144</end></extent><extent unit="issue pages"><start>A4</start><end>A48</end></extent><extent unit="issue pages"><start>A49</start><end>A60</end></extent><extent unit="issue pages"><start>A61</start><end>A72</end></extent><extent unit="issue pages"><start>A73</start><end>A86</end></extent><extent unit="issue pages"><start>A87</start><end>A100</end></extent><extent unit="issue pages"><start>525</start><end>790</end></extent><extent unit="pages"><start>562</start><end>569</end></extent></part></relatedItem><identifier type="istex">2ABF2798E1258AFD14D8AAA399E1A624D5EFBF91</identifier><identifier type="DOI">10.1016/0002-9149(81)90539-7</identifier><identifier type="PII">0002-9149(81)90539-7</identifier><identifier type="ArticleID">81905397</identifier><recordInfo><recordContentSource>ELSEVIER</recordContentSource></recordInfo></mods></metadata><enrichments><istex:refBibTEI 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